decreased libido in humans (because testosterone could not be formed);
suppression of the immune response due to the deficiency of vitamin D, (which is produced precisely by cholesterol), followed by depression;
poor control of the immune system, as a cholesterol deficiency also causes a decrease in blood cortisol;
a decrease in female estradiol with serious consequences, such as osteoporosis.
There is a fundamental concept to point out: the intake of food cholesterol does not negatively influence the cholesterol present in our arteries. In fact, it helps eliminate it. Our bodies are equipped with an internal control mechanism for the production of cholesterol. As soon as we take in cholesterol in the form of food, the liver itself no longer produces it, meaning that the amount taken in will never ever increase the amount already present in the blood. Had it not been so, the French, with all the cholesterol they ingest every day, would have died out from all the heart attacks. More than anything else, we need to understand why the body produces high amounts of cholesterol. Could it be inflammation, given how cholesterol is needed to restore cell membranes? Perhaps it is poor sex hormone formation, seeing as cholesterol is used for this as well? Is it a need for cortisol production, an anti-inflammatory molecule derived from cholesterol? But then how does atherosclerotic plaque form?
Recent studies have shown that the formation of atherosclerotic plaque is the final stage of a path that begins with the alteration of blood vessels due to the constant presence of high blood sugar levels. In other words, when the structure of a blood vessel is altered, cholesterol gets into the endothelial tissue of the vessel, with subsequent inflammation that entails the deposit of platelets and the formation of atherosclerotic plaque. Furthermore, recent studies cite that the infiltration of cholesterol into blood vessel tissue depends both on the size of the LDL cholesterol (known as bad cholesterol) and on the degree of oxidation of these small and oxidized LDL particles that penetrate the blood vessel. On the contrary, large and non-oxidized LDLs do not have this capability. As such, checking the LDL values in tests is pertinent, since only very few medical testing centers evaluate the oxidation and size of LDL particles. Another value to check is the ratio between HDL (known as good cholesterol) and total cholesterol. My total cholesterol could be above 200, but if the HDL ratio is lower than 4, I would not have much cause for concern, as I would consider myself pretty protected. I have seen patients with 220 total cholesterol, and 120 HDL cholesterol, who came to me frightened enough to start an anti-cholesterol diet because they were threatened by the imminent prescription of statins because their cholesterol was out of bounds! I can imagine the day will come when all of mankind has statins for breakfast Crazy stuff! Statins are drugs with numerous side effects, which are described in the leaflet accompanying the aforementioned drug: joint pain, severe peeling of the skin; blisters of the skin, mouth, genitals and eyes, and liver disorders, among many others.
As you can see, these are problems that are almost always dealt with via other medications, not realizing that it may be the medication itself causing the ailments. The leaflet also states that these medications must be used only in the event that proper diet and physical activity fail. I can tell you from personal experience that half the patients who come to my office take statins without even having tried the dietary route because they thought their cholesterol levels were genetic. Doctor, I have it because my mom did, and my grandmother before her When I hear this, my answer is as follows: Dear patient, you have it because you are eating what your mother, grandmother, and predecessors ate. Guess what the average Italian eats? Pasta, bread, potatoes, pizza, and all the other crap made of white flour and sugar! Now, you must be thinking: But what does pasta have to do with cholesterol? It has been known for years that blood cholesterol levels rise not because cholesterol is introduced by way of food, but because of a diet rich in flours, especially white flour and sugar. In short, the typical diet of the Western population, who are great experts in chemistry, biochemistry, medicine, and engineering, and who are even capable of reaching the moon, have received very little education when it comes to food science, which should be one of the main subjects taught to everyone starting in elementary school, in the name of prevention!
Today, we know that saturated fats are not the real culprits of atherosclerosis. As mentioned above, plaque does not form if an artery is not damaged, and it occurs only following continuous inflammation due to high levels of glycemia and insulin; in fact, atherosclerotic plaques are lifelong companions of diabetics. It is not a coincidence!
The reason we are afraid of saturated fats stems from the fact that, in 1967, a few scientists were paid by the sugar industry to write up false studies whereby the important role sugar plays in the genesis of atherosclerosis was omitted. These studies claimed that fats caused all cardiovascular disorders. For years, professionals have believedand many continue to believein these studies, referring back to them whenever anyone makes the counterclaim that the cause of cardiovascular damage is actually sugars and not fats.
A series of studies done in France in 1979 has negated the link between the intake of saturated fats/cholesterol and cardiovascular diseases, leading to what is known today as the French paradox. These studies showed that the French, who are egg and cheese eaters, died less from cardiovascular diseases compared to other European countries. The scientists, who supported the false theory surrounding fats, interpreted this phenomenon as stemming from the heavy consumption of red wine. And so the beautiful fairy tale claiming wine is good for the arteries was born. It was purportedly thanks to resveratrol, (the flavonoid compound in wine), and its protective cardiovascular effects. Too bad that, in order to make use of such an effect, you would need to consume one liter of red wine a day, which would result in serious liver damage. At the time, they did not understand that the French, though large consumers of saturated fats, did not consume the same quantities of white flours and sugars as Italians, Americans and most other Europeans.
Finally, in September 2017, in an important conference on cardiology held in Barcelona, scientists presented a series of studies that actually showed that saturated fats could actually prevent strokes.
Saturated fats, in general, also promote hormonal production. In addition, some of them are transformed into other, less dangerous fats as soon as they enter our bodies. For example, the stearic acid found in cocoa is converted into oleic acid, a beneficial monounsaturated fat abundantly present in olive oil. Stearic acid is not dangerous at all because it is easily incorporated into our fat mass, which is entirely made up of stearic acid. Another example is palmitic acid, found in palm oil, which is also converted into stearic acid. Yes, you read that correctly! In reality, the supposedly horrible palm oil poses no danger whatsoever to our cardiovascular system, precisely because the biochemical pathway that carries palmitic acid leads to its transformation into stearic acid. Before pointing to palm oil as the menace responsible for atherosclerosis, I would examine the products in which it is contained. Do you know which products contain it? They are in creamy sweets full of sugar, glucose syrup, fructose and other sweetenersall of the primary things responsible for cardiovascular diseases! These days, on all the packaging for cookies, sweets, and other products put out by the confectionery industry, you will find the wording: Does not contain palm oil, as if to mean without any health risk.